Biomedicines | Free Full-Text | Targeting NF-κB Signaling by Calebin A, a Compound of Turmeric, in Multicellular Tumor Microenvironment: Potential Role of Apoptosis Induction in CRC Cells
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Nuclear factor kappa-B and the heart - ScienceDirect
Frontiers | Nuclear Factor κB (NF-κB)–Mediated Inflammation in Multiple Sclerosis
NF-κB, inflammation, immunity and cancer: coming of age | Nature Reviews Immunology
Blocking Nuclear Factor-Kappa B Protects against Diet-Induced Hepatic Steatosis and Insulin Resistance in Mice | PLOS ONE
NF‐κB signaling in inflammation and cancer - Zhang - 2021 - MedComm - Wiley Online Library
Cells | Free Full-Text | Roles of NF-κB in Cancer and Inflammatory Diseases and Their Therapeutic Approaches
30 Years of NF-κB: A Blossoming of Relevance to Human Pathobiology: Cell
Canonical and noncanonical NF-κB signaling pathways. The canonical... | Download Scientific Diagram
Frontiers | Nuclear Factor-κB Dysregulation and α-Synuclein Pathology: Critical Interplay in the Pathogenesis of Parkinson's Disease
NF-κB-related genetic diseases | Cell Death & Differentiation
Gentiopicroside activates the bile acid receptor Gpbar1 (TGR5) to repress NF-kappaB pathway and ameliorate diabetic nephropathy - ScienceDirect
Cells | Free Full-Text | Inhibitory-κB Kinase (IKK) α and Nuclear Factor-κB (NFκB)-Inducing Kinase (NIK) as Anti-Cancer Drug Targets
Nuclear Factor-κB — A Pivotal Transcription Factor in Chronic Inflammatory Diseases | NEJM
Cells | Free Full-Text | The Ubiquitination of NF-κB Subunits in the Control of Transcription
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Blocking Nuclear Factor-Kappa B Protects against Diet-Induced Hepatic Steatosis and Insulin Resistance in Mice | PLOS ONE
Blocking Nuclear Factor-Kappa B Protects against Diet-Induced Hepatic Steatosis and Insulin Resistance in Mice | PLOS ONE
Blocking Nuclear Factor-Kappa B Protects against Diet-Induced Hepatic Steatosis and Insulin Resistance in Mice | PLOS ONE
Frontiers | Transforming Growth Factor-Beta (TGF-β) Signaling in Cancer-A Betrayal Within
Frontiers | NFκB and Kidney Injury
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